Canis ISSN: 2398-2942


Contributor(s): Richard Walker




  • Family: Pseudomonadaceae.
  • Genus: Pseudomonas.

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Clinical Effects



  • Mainly saprophytic.


  • Multiply in soil, water and feces. Can multiply in the host if normal flora have been disrupted.


  • Environmental or endogenous exposure is constant. Most infections are secondary to compromised host defences.

Pathological effects

  • Capable of causing various diseases in wide range species.
  • Disease often due to production of toxins.
  • Dogs - most commonly associated with otitis externa and cystitis.
  • Horses - cause of corneal ulcers and uterine infection.
  • Cows - mastitis.

Other Host Effects

  • Predisposing factors allow opportunist infection withPseudomonas, eg burns, reduction in normal flora following antibiotic treatment.


Control via chemotherapies

  • Some species, eg P. aeruginosa Pseudomonas aeruginosa have multiple drug resistance.
  • Often susceptible to gentamycin, tobramycin, amikacin and carbenicillin for the treatment of soft tissue infections.
  • Tetracycline can achieve adequate concentrations in the canine urinary tract to kill most isolates.
  • Usually susceptible to the levels of antimicrobials present in otic preparations.


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Further Reading


Refereed papers

  • Recent references from PubMed and VetMedResource.
  • Nuttall T J (1998) Use of ticarcillin in the management of otitis externa complicated by Pseudomonas aeruginosa. JSAP 39 (4), 165-168 PubMed.
  • Willard M D, Berridge B, Braniecki A et al (1998) Possible antibiotic-associated colitis in a dog. JAVMA 213 (12), 1775-1779, 1753-1754 PubMed.
  • Dahlinger J, Marks S L & Hirsh D C (1997) Prevalence and identity of translocating bacteria in healthy dogs. JVIM 11 (6), 319-322 PubMed.
  • Court E A, Watson A D & Martin P (1994) Pseudomonas aeruginosa bacteremia in a dog. Australian Vet J 71 (1), 25-27 PubMed.