ISSN 2398-2942      

Mycoplasma haemocanis

icanis
Contributor(s):

Richard Walker

Synonym(s): Hemobartonella canis, H. canis


Introduction

Classification

Taxonomy

  • Rickettsia.
  • Family: Mycoplasmataceae.
  • Genus: Mycoplasma.

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Clinical Effects

Epidemiology

Habitat

  • Attaches to canine erythrocyte membranes.
  • Host species-specific.
  • Ticks may be reservoir hosts.

Lifecycle

  • Reproduces in inclusion bodies attached to erythrocyte membrane.
  • It is possible that multiplication may occur in ticks.

Transmission

  • Ticks Ixodes ricinus.
  • Blood transfusion.
  • In utero.
  • Most studies of transmission mechanisms have been experimental.

Pathological effects

  • Disease is caused by immune-mediated destruction of eythrocytes.
  • Immunosuppression potentiates the severity of the disease.

Other Host Effects

  • May be an incidental finding on blood smears.
  • Often non-pathogenic.

Control

Control via chemotherapies

  • Tetracyclines Tetracycline are the treatment of choice.
  • May require concurrent glucorticoid therapy to stop immune-mediated destruction of erythrocytes.
  • Chlorpromazine or metronidazole may be required for resistant strains.

Diagnosis

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Further Reading

Publications

Refereed papers

  • Recent references from PubMed and VetMedResource.
  • Brinson J J & Messick J B (2001) Use of a polymerase chain reaction​ assay for detection of Haemobartonella canis in a dog. JAVMA 218 (12), 1943-1945, 1936 PubMed.
  • Lester S J, Hume J B & Phipps B (1995) Haemobartonella canis infection following splenectomy and transfusion. Can Vet J 36 (7), 444-445 PubMed.
  • Gretillat S (1981) Haemobartonella canis (Kiküth 1928) in the blood of dogs with parvovirus disease. JSAP 22 (10), 647-653 PubMed.
  • Pryor W H Jr. & Bradbury R P (1975) Haemobartonella canis infection in research dogs. Lab Anim Sci 25 (5), 566-569 PubMed.

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